Semax and BDNF Pathway Activity in Preclinical Neuroscience
What is Semax?
Semax is a synthetic heptapeptide developed in Russia, derived from the 4–7 fragment of adrenocorticotropic hormone (ACTH) with an added Pro-Gly-Pro tail that improves its stability. Unlike full ACTH, the ACTH(4–7) fragment is studied for its neurological signalling properties without the hormonal (corticosteroid-releasing) activity of the parent molecule.
Semax comes from a substantial Eastern European research tradition and is studied as a neuropeptide research reagent — particularly for its association with neurotrophic signalling in the brain.
BDNF, NGF and neuroprotection
Neurotrophic factor expression. The most studied property of Semax in preclinical models is its association with increased expression of brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) — signalling proteins central to neuron survival, growth and synaptic plasticity. Rodent studies have examined changes in BDNF/NGF levels and downstream TrkB-receptor signalling following Semax administration.
Neuroprotection models. Semax has been studied in rodent models of cerebral ischaemia and oxidative stress, where research has examined its association with neuronal survival and modulation of inflammatory and antioxidant gene expression.
Cognitive and attention pathways. Preclinical work has also looked at Semax in relation to attention, learning and the dopaminergic and serotonergic systems, making it a research tool for studying neuropeptide modulation of cognition.
Semax and Selank
Semax is frequently studied alongside Selank, another Russian-developed neuropeptide. The two are often paired in cognitive and neuropeptide research because they are associated with complementary pathways — Semax with neurotrophic (BDNF/NGF) signalling and Selank with GABAergic modulation. Both sit within the neuropeptide research category.
Velox Peptides supply information
Velox Peptides supplies Semax as a lyophilised powder at ≥99.0% HPLC-verified purity with a batch certificate of analysis available on request. For reconstitution, see the reconstitution calculator. Supplied strictly as a research reagent for in vitro use.
References & further reading
- Dolotov OV et al. “Semax, an analog of ACTH(4-7), regulates expression of brain-derived neurotrophic factor.” Journal of Neurochemistry, 2006.
- Ashmarin IP et al. “Semax in the prevention of disease and the maintenance of working capacity.” Neuroscience and Behavioral Physiology, review literature.
Summaries are paraphrased from the peer-reviewed preclinical literature. For full source citations, email veloxpeps@gmail.com.